General Pediatrics Board Exam Prep
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The ABP 2022 General Pediatrics Pass Rate for the Certification exam is 80%.
Note: The ABP did not release the 2022 pass rates for the Maintenance of Certification exam.
Compare to exam takers who prepared with The Pass Machine:
In 2022, The Pass Machine Pediatrics Board Review clients achieved a 100% pass rate on the MOC exam!
1 in 5 Failed the Pediatrics Cert
The Pediatrics Initial Certification Exam is a relatively challenging of board exam. Is 2022, 678 out of 3,364 first-time takers didn’t pass the Pediatrics Cer exam.
ABP Pass Rate Deep Dive
For granular APB pass rate data, check out ABP pass rates by residency program for American Board of Pediatrics 2020-2022 Pediatric Training Program Pass Rates
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What do I do if I fail the Pediatrics board exam?
The first thing you should do is just take it easy, sleep on it. Give yourself a few days, a week or two to come to terms with what has happened. Your next exam is six months to a year away. Dr. Jack has more advice in this video, What Do You Do If You Fail Your Medical Board Exam?
Pediatrics Certification Exam Scoring
Pediatric Critical Care exam results are reported using a 1 to 300 scale, with 180 designated as the passing mark. A test taker’s score on the 1 to 300 scale does not reflect the percentage of questions answered correctly. Instead, a 180 represents the minimum level of knowledge required to pass the examination.
Pediatrics Exam Format
Pediatric Critical Care Exam questions are written in a single-best-answer multiple-choice format. Each question is followed by four or five answer options, one of which is the correct answer. Questions may require the interpretation of a graphical illustration, such as x-ray studies, growth charts, and photographs, to answer correctly.
Example of a single-best-answer multiple-choice question format:
A newborn baby is born to a 32-year-old mother. He was born at 39 weeks of gestation via Cesarean section. The birth weight is 8 pounds, 15 ounces, and macrosomia is noted. The APGAR score was 4 at 1 minute and 9 at 5 minutes. The mother has a previous history of rheumatoid arthritis, for which she takes methotrexate. However, the rheumatologist asked her to stop taking the medication during the pregnancy. She does not have a history of hypertension, diabetes mellitus, or any other medical conditions. Physical examination reveals a BMI of 31 kg/m2. The pregnancy has remained uneventful, apart from 2 urinary tract infections in the first trimester and one episode in the second trimester. What is the most likely mechanism for macrosomia in this patient?
◯ A. Elevated fetal phenylalanine levels
◯ B. Enhanced fetal nutrition
◯ C. Fetal hyperglycemia
◯ D. Not known
◯ E. Nutritional programming
D. Not known is correct because the mechanism of macrosomia in this child is unknown. Other than an increased BMI that falls in the range of obese, the mother does not have a history of any medical condition or risk factor that is likely to result in macrosomia. Obesity is linked to macrosomia and hypoglycemia in newborn babies, but the mechanism for macrosomia in mothers with obesity is still to be determined.
Elevated fetal phenylalanine levels is incorrect because there is nothing in the history to suggest a problem with phenylalanine levels. Babies of mothers who have phenylketonuria have elevated fetal phenylalanine values. These elevated values can cause microcephaly and retardation.
Enhanced fetal nutrition is incorrect because that does not seem to be the likely cause of macrosomia in this baby. Enhanced fetal nutrition would ensure sufficient and uniform fetal development rather than an imbalance between the development of the head and the rest of the body causing macrosomia.
Fetal hyperglycemia is incorrect because that seems unlikely in this patient. The mother does not have a history of diabetes mellitus. Furthermore, babies of mothers having diabetes mellitus are usually large for gestational age.
Nutritional programming is incorrect because that is not a mechanism that results in macrosomia. Poor maternal nutrition results in reduced fetal nutrients and nutritional programming. This, in turn, causes intrauterine growth restriction and adult insulin resistance.
Kliegman R, Stanton B, St. Geme J, Schor N. Nelson Textbook of Pediatrics. 20th ed. Elsevier; 2016:804.
Fetal and Neonatal Care